📄 Certificate of Analysis available on request
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Matrix-4 Research Set is supplied as a set of laboratory research compounds with defined reference identities. The set contains CJC-1295 + Ipamorelin, TB-500, BPC-157, each in its own vial.
This listing is limited to product identity, analytical documentation, and research-use status. Each component is provided for qualified laboratory research only and is not intended for human or veterinary use. Each batch is independently tested for identity, purity, and consistency, with a lot-specific Certificate of Analysis provided for documentation and internal research records.
Each compound in this set has been characterised in preclinical laboratory models. The primary studies below, in animal and in-vitro models, examined cellular, biochemical, and mechanistic endpoints. The literature is preclinical: it describes the compounds, not any outcome in humans.
Animal modelHuman growth hormone-releasing factor (hGRF)1-29-albumin bioconjugates activate the GRF receptor on the anterior pituitary in rats: identification of CJC-1295 as a long-lasting GRF analogIn cultured rat anterior-pituitary cells and rats, the albumin-bioconjugated analog activated the GRF receptor and extended plasma residence.Read the study ↗
Animal modelIpamorelin, the first selective growth hormone secretagogueIn primary rat pituitary cells, rats, and swine, the peptide released growth hormone via a GHRP-like receptor with selectivity for GH.Read the study ↗
In vitroThe actin binding site on thymosin beta4 promotes angiogenesisIn an in-vitro endothelial and chick aortic-ring model, the actin-binding motif of the peptide was mapped as the structural determinant of endothelial adhesion and vessel-sprouting endpoints.Read the study ↗
In vitroThymosin beta4 induces endothelial progenitor cell migration via PI3K/Akt/eNOS signal transduction pathwayIn cultured endothelial progenitor cells, the peptide was assayed for dose-dependent cell-migration endpoints, with a proposed mechanism via PI3K/Akt/eNOS signal transduction.Read the study ↗
In vitroPentadecapeptide BPC 157 enhances the growth hormone receptor expression in tendon fibroblastsIn cultured rat tendon fibroblasts, the peptide was studied for growth hormone receptor expression at the mRNA and protein level, with a proposed mechanism via JAK2 signal transduction.Read the study ↗
Animal modelModulatory effects of BPC 157 on vasomotor tone and the activation of Src-Caveolin-1-endothelial nitric oxide synthase pathwayIn isolated rat thoracic aorta, the peptide was studied for endothelium-dependent vasomotor-tone endpoints, with a proposed mechanism via the Src-Caveolin-1-eNOS pathway.Read the study ↗
Full documentation, on the record. A Certificate of Analysis and a Safety Data Sheet are available on request for every batch.
Links open the original study on PubMed. For research and educational purposes, descriptive of the published preclinical literature, not therapeutic claims about any ai-peptides product.
